期刊簡介預計審稿時間： 12 Weeks

The only open access journal in its field, Skeletal Muscle publishes novel, cutting-edge research and technological advancements that investigate the molecular mechanisms underlying the biology of skeletal muscle. Reflecting the breadth of research in this area, the journal welcomes manuscripts about the development, metabolism, the regulation of mass and function, aging, degeneration, dystrophy and regeneration of skeletal muscle, with an emphasis on understanding adult skeletal muscle, its maintenance, and its interactions with non-muscle cell types and regulatory modulators.

Main areas of interest include:

-differentiation of skeletal muscle-

atrophy and hypertrophy of skeletal muscle-

aging of skeletal muscle-

regeneration and degeneration of skeletal muscle-

biology of satellite and satellite-like cells-

dystrophic degeneration of skeletal muscle-

energy and glucose homeostasis in skeletal muscle-

non-dystrophic genetic diseases of skeletal muscle, such as Spinal Muscular Atrophy and myopathies-

maintenance of neuromuscular junctions-

roles of ryanodine receptors and calcium signaling in skeletal muscle-

roles of nuclear receptors in skeletal muscle-

roles of GPCRs and GPCR signaling in skeletal muscle-

other relevant aspects of skeletal muscle biology.

In addition, articles on translational clinical studies that address molecular and cellular mechanisms of skeletal muscle will be published. Case reports are also encouraged for submission.

Skeletal Muscle reflects the breadth of research on skeletal muscle and bridges gaps between diverse areas of science for example cardiac cell biology and neurobiology, which share common features with respect to cell differentiation, excitatory membranes, cell-cell communication, and maintenance. Suitable articles are model and mechanism-driven, and apply statistical principles where appropriate; purely descriptive studies are of lesser interest.

《骨骼肌》是該領域唯一的開放獲取期刊，發(fā)表研究骨骼肌生物學背后分子機制的新穎、尖端研究和技術進步。為反映該領域研究的廣度，該期刊歡迎有關骨骼肌的發(fā)育、代謝、質量和功能調節(jié)、衰老、退化、營養(yǎng)不良和再生的稿件，重點是了解成人骨骼肌、其維持及其與非肌肉細胞類型和調節(jié)劑的相互作用。

主要感興趣的領域包括：

-骨骼肌分化-

骨骼肌萎縮和肥大-

骨骼肌衰老-

骨骼肌再生和退化-

衛(wèi)星細胞和衛(wèi)星樣細胞的生物學-

骨骼肌營養(yǎng)不良性退化-

骨骼肌中的能量和葡萄糖穩(wěn)態(tài)-

非營養(yǎng)不良性遺傳疾病骨骼肌，如脊髓性肌萎縮和肌病-

神經肌肉接頭的維持-

瑞安諾丁受體和鈣信號在骨骼肌中的作用-

核受體在骨骼肌中的作用-

GPCR 和 GPCR 信號在骨骼肌中的作用-

骨骼肌生物學的其他相關方面。

此外，還將發(fā)表關于骨骼肌分子和細胞機制的轉化臨床研究文章。也鼓勵提交病例報告。

骨骼肌反映了骨骼肌研究的廣度，并彌合了不同科學領域之間的差距，例如心臟細胞生物學和神經生物學，它們在細胞分化、興奮膜、細胞間通訊和維持方面具有共同特征。合適的文章是模型和機制驅動的，并在適當?shù)牡胤綉媒y(tǒng)計原理；純粹的描述性研究不太有趣。

《Skeletal Muscle》(骨骼肌)編輯部通訊方式為CAMPUS, 4 CRINAN ST, LONDON, ENGLAND, N1 9XW。如果您需要協(xié)助投稿或潤稿服務，您可以咨詢我們的客服老師。我們專注于期刊咨詢服務十年，熟悉發(fā)表政策，可為您提供一對一投稿指導，避免您在投稿時頻繁碰壁，節(jié)省您的寶貴時間，有效提升發(fā)表機率，確保SCI檢索（檢索不了全額退款）。我們視信譽為生命，多方面確保文章安全保密，在任何情況下都不會泄露您的個人信息或稿件內容。